Obese people, the content of not less than normal, although some even higher than normal, due to reduced insulin receptor on the cell or receptor defects, the role of insulin resistance, and the consumption of fat cells in vivo glucose is the major places. 
As we all know, obesity is one of the important factors for type 2 diabetes. In the long term obese population, the prevalence of diabetes increased significantly, than the general population prevalence of up to four times as much. On the other hand, in type 2 diabetic patients, 80 percent are obese. 
Obesity and diabetes everyone because everyone fruit? 
      Development of obesity have trilogy, namely obesity, metabolic syndrome, cardiovascular and cerebrovascular diseases. One of metabolic syndrome, obesity is caused by diabetes. So in the end lead to diabetes is obesity? Obesity or diabetes caused it? 
Diabetes experts explained, it should be said that obese type 2 diabetes, metabolic blood pressure, gout, dyslipidemia and obesity are closely related etc, can be regarded as complications of obesity. Complications, treatment of complications caused by obesity as standard, cure obesity-oriented. Without treatment of the primary disease, hypoglycemic drugs alone, is a palliative. 
      For type 2 diabetes, obesity and obesity, experts point out that obesity is like a huge iceberg at sea, diabetes, hypertension, dyslipidemia, gout, etc. are just the tip of the iceberg exposed sea level. Eliminating the tip of the iceberg, only a temporary solution, eliminating the ice lump side to cure. 
      Obesity may lead to different diseases produce 
      Mentioned above, obesity, Type 2 diabetes can be said that obesity complications. We can understand the complications of obesity leads into two categories: one is associated with metabolic complications, such as metabolic hypertension, dyslipidemia, fatty liver, diabetes, gout, etc; brought by a class of non-overweight metabolic diseases, such as bone and joint damage, cervical spondylosis, lumbar, lumbar disc herniation, knee aging. 
      Abdominal obesity can be divided into non-abdominal two outcomes are not the same. Higher probability of formation of abdominal obesity metabolic syndrome, rather than a non-metabolic complications of abdominal often occurs. After a large number of domestic and foreign researchers in clinical practice, epidemiology, etc., have established a variety of obesity measurements, such as body mass index (BMI) and so on. The BMI's shortcomings is that right abdomen, non-abdominal not be divided subtypes. In other words, the same abdominal obesity also have higher levels of harm, and some of the relatively low degree of harm. Professor playing with a very vivid metaphor, like stuffing a large thick-skinned type of intra-abdominal fat and more abdominal fat, thick and more than a relatively large body of subcutaneous fat, the risk would be smaller; kind of thin-skinned and big stuffing type of intra-abdominal fat and more abdominal fat, thin, like spider-Man, like tiny limbs, big belly, harm will be much larger. 
      So, just from the body mass index is not objectively reflect more with less subcutaneous fat of. Because BMI great two situations: one is overweight body lean body tissue, but not excessive amount of body fat, BMI overweight or obese this situation, but may not be harmful, but muscular; another is excess body fat, which is the true obesity. And sometimes even normal BMI, body fat may be excessive. 
      Why do fat people susceptible to diabetes? 
      Fat people due to excessive calorie intake, consumption is reduced, resulting in long-term nutrition, obesity prone to diabetes is the real reason. In addition, obese people susceptible to diabetes is the key: insulin content of fat in humans, although not lower than normal and some even higher than normal, due to the reduction of insulin receptors on cells or receptor defects, resistance to insulin action , while consumption of fat cells in vivo glucose is the main place. 
      In order to reduce the time, in the obese state, the body would have to enhance the pancreatic p cells insulin secretion, to counteract against the role of fat, particularly obese people, insulin reserve function as soon as the growth of less fat, insulin is produced relatively low. In addition, fatty degeneration of the liver damage is reduced so use insulin intake and excessive oxygen free radicals, the relative lack of SOD scavenging ability, have led to obesity than others prone to disease.